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Patients with a history of immunosuppression should be tested for progressive multifocal leukoencephalopathy by cerebrospinal fluid (CSF) examination for the presence of JC virus. A high index of suspicion should remain for neoplasia, including primary central nervous system (CNS) lymphoma and gliomatosis cerebri.
Its pathophysiology is speculated to resemble delayed post-hypoxic leukoenceph-alopathy (DPHL), which involves mild to moderate hypoxic damage of oligodendrocytes [3]. One previous report has shown association with Libman–Sacks endocarditis, sug-gesting a possible role of autoimmune inflammation [1].
28 sie 2020 · Statistical analysis. We analyzed the following outcomes: demographic profiles, baseline SBP, baseline NIHSS score, pre-existing risk factors, stroke mechanism, tangible thrombolysis, and main clinical outcomes. Outcome meta-analysis was conducted using the random-effect model.
26 gru 2022 · Clinical symptoms suggestive of delayed post-hypoxic leukoencephalopathy (DPHL) can be observed in very rare cases following successful revascularization therapy in large vessel occlusions, causing acute ischemic stroke.
22 lis 2019 · Given the success in preventing stroke in general, with important reductions in incidence and mortality, are new treatments to target cSVD progression even necessary? That they are is supported by the evidence that many patients with cSVD experience progression with cognitive and functional decline despite conventional stroke risk reduction.
Post-ischemic Leukoencephalopathy after Endovascular Treatment for Acute Ischemic Stroke. Can J Neurol Sci. 2019 May;46 (3):363-365. doi: 10.1017/cjn.2019.30.
Background: Delayed post-ischemic leukoencephalopathy (DPIL) is a rare complication after mechanical thrombectomy, with no well-established clinical characteristics and pathomechanism. We explored the characteristics and possible mechanisms in three pa-tients with DPIL.
