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Cutaneous LE (CLE) manifests with a wide range of skin lesions divided into acute, subacute and chronic subtypes. Despite classic forms of CLE, such as malar rash or discoid LE, little-known variants may occur, for instance hypertrophic LE, chilblain LE and lupus panniculitis.
19 maj 2021 · Patients with ACLE, bullous lupus, and nonspecific cutaneous lesions of lupus (e.g., vasculopathic lesions, see Figure 1) all have a higher risk of developing systemic lupus when compared to individuals diagnosed with SCLE, DLE, lupus tumidus, lupus panniculitis, or chilblain lupus [1].
Skin lesions in patients with lupus may be specific or nonspecific. This paper covers the SLE-specific cutaneous changes: malar rash, discoid rash, photosensitivity, and oral mucosal lesions as well as SLE nonspecific skin manifestations, their pathophysiology, and management.
7 sie 2024 · By leveraging genetic variants as proxies for HPV infection and performing MR analysis, we aimed to provide clearer insights into whether HPV contributes to the development of SLE or vice versa.
7 sie 2024 · In this study, we employed a bidirectional two-sample Mendelian randomization (MR) approach to investigate the potential causal relationship between human papillomavirus (HPV) infection and systemic lupus erythematosus (SLE).
CLE patients display well-defined skin lesions, often in sun-exposed areas. The disease often has a chronic and relapsing course that can be induced or aggravated by UV light.
Skin lesions caused by HPV are diverse and may include warts, flat warts, epidermodysplasia verruciformis (EV), and precancerous and cancerous lesions. Warts are the most common skin lesions associated with HPV and usually appear on the hands, feet, and other areas of the body.
