Search results
17 lip 2023 · Falling blood pressure does the opposite: dilate the afferent arteriole and preserve blood flow to the kidney. Secondly, the renin-angiotensin-aldosterone system acts to preserve GFR. The juxtaglomerular cells in the afferent arteriole release renin in response to decreased stretch.
15 maj 2018 · An increase in the afferent arteriolar diameter (decrease in resistance) causes an increase in the glomerular capillary hydrostatic pressure and an increase in GFR. A decrease in the diameter of the afferent arteriole has the opposite effect.
The efferent arterioles form a convergence of the capillaries of the glomerulus, and carry blood away from the glomerulus that has already been filtered. They play an important role in maintaining the glomerular filtration rate despite fluctuations in blood pressure.
Vasodilation in the afferent arteriole and vasconstriction in the efferent arteriole will increase blood flow (and hydrostatic pressure) in the glomerulus and will increase GFR. Conversely, vasoconstriction in the afferent arteriole and vasodilation in the efferent arteriole will decrease GFR.
17 lip 2023 · The kidneys receive 20% to 25% of the cardiac output (about 1.0 to 1.1 liters per minute) with the blood entering individual glomerular tufts via the afferent arteriole and exiting through the efferent arteriole. Of this renal blood flow (RBF), only the plasma can cross the structures comprising the glomerulus.
1 maj 2024 · Renal arteriole endothelial cells produce a number of locally acting hormones (autacoids). Endothelin is a powerful vasoconstrictor of both afferent and efferent arterioles leading to a reduction in both RPF and GFR. Nitric oxide is a potent vasodilator leading to increases in RPF and GFR.
28 lip 2016 · In fact, a large family of drugs is capable of bringing about an increase in hydrostatic pressure gradient along the efferent arteriole of the kidney, generated either by an increased resistance in, or a higher blood flow through, these vessels.
