Search results
8 paź 2024 · The ECG features of digoxin effect are seen with therapeutic doses of digoxin and are due to: Shortening of the atrial and ventricular refractory periods — producing a short QT interval with secondary repolarisation abnormalities affecting the ST segments, T waves and U waves. Increased vagal effects at the AV node — causing a prolonged PR interval
- Ed Burns
Emergency Physician in Prehospital and Retrieval Medicine in...
- ECG Library
ECG Library Content. ECG Basics – Waves, Intervals, Segments...
- Digoxin Toxicity
ECG Features of Digoxin Toxicity. The classic digoxin toxic...
- Ed Burns
8 paź 2024 · ECG Features of Digoxin Toxicity. The classic digoxin toxic dysrhythmia combines: Supraventricular tachycardia (due to increased automaticity) Slow ventricular response (due to decreased AV conduction) Other common dysrhythmias associated with digoxin toxicity include:
OVERVIEW. Digoxin toxicity is characterised by gastrointestinal distress, hyperkalemia and life-threatening dysryhthmias, including increased automaticity and AV nodal blockade. Digoxin has a narrow therapeutic index and chronic toxicity is more likely in the elderly and those with renal impairment. Chronic digoxin toxicity varies in severity ...
Digoxin ECG changes: arrhythmias, conduction defects and waveform changes. Digoxin may be used in patients with heart failure, atrial fibrillation, atrial flutter, and in selected cases of paroxysmal supraventricular tachycardia.
The diagnosis of digoxin toxicity is based primarily on clinical suspicion and clinical features (including ECG changes) suggestive of digoxin intoxication, and can be confirmed by assessment of serum digoxin levels.
10 lut 2020 · Electrocardiography (ECG) is an important way to evaluate patients with cirrhosis and may reveal significant changes associated with liver disease. Our study aimed to evaluate ECG changes in patients with diagnosed liver cirrhosis and compare them to patients with chronic hepatitis.
8 sie 2024 · Digoxin toxicity is a clinical diagnosis that relies in part on ECG findings such as signs of increased automaticity and atrioventricular node blockade (premature ventricular contractions, slowed ventricular response), but also on clinical features, history of digoxin intake, history of other illnesses, and elevated digoxin concentrations.
