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This article concerns adverse side effects of digoxin treatment, with emphasis on ECG changes, arrhythmias and conduction disturbances. Digoxin may cause all known cardiac arrhythmias.
- Atrioventricular
1 st degree AV block; 2 nd degree AV block; 3 rd degree AV...
- Atrioventricular
8 paź 2024 · The ECG features of digoxin effect are seen with therapeutic doses of digoxin and are due to: Shortening of the atrial and ventricular refractory periods — producing a short QT interval with secondary repolarisation abnormalities affecting the ST segments, T waves and U waves. Increased vagal effects at the AV node — causing a prolonged PR interval
In addition to the arrhythmias considered above, changes on the ECG typical of digoxin toxicity can include down-sloping ST segment depression (‘scooped out’ or ‘reverse tick’ appearance), flattened/inverted T waves and increased U wave amplitude.
8 paź 2024 · Digoxin can cause a multitude of dysrhythmias due to: Increased automaticity (increased intracellular calcium) Decreased AV conduction (increased vagal effects at the AV node) Clinical features of Digoxin Toxicity. GIT: Nausea, vomiting, anorexia, diarrhoea. Visual: Blurred vision, yellow/green discolouration, haloes.
1 sty 2020 · Digoxin is a commonly prescribed drug in the management of heart failure and atrial fibrillation. Despite its widespread use, most clinicians have little experience with recognising clinical signs and symptoms that might suggest a potentially lethal drug toxicity.
ECG showed sagging ST segment, biphasic T wave and atrial fibrillation with slow ventricular response. Laboratory/imaging work ups, including cerebrospinal fluid analysis, were unrevealing. Digoxin level was checked, showing 2.2 ng/ml.
Patients should have continuous ECG, temperature, BP and potassium concentration monitoring during and for at least 24 hours after administration. Interactions May interfere with digoxin immunoassay measurements.
