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  1. 30 maj 2024 · We will examine the epidemiology of OSA and its association with various cardiovascular conditions, explore the underlying pathophysiological mechanisms linking OSA to cardiovascular disease, discuss the clinical implications of OSA-related cardiovascular complications, and review current approaches to diagnosing and managing cardiovascular ...

  2. 11 maj 2024 · Recent studies demonstrate a link between SDB and HF. SDB encompasses obstructive sleep apnea (OSA) and central sleep apnea (CSA) and is common in HF patients, affecting 50–80%, 47–76% in patients who have HF with reduced ejection fraction (HFrEF) and 55% in HF with preserved ejection fraction (HFpEF). . The Sleep-Disordered Breathing in ...

  3. 28 cze 2019 · Central sleep apneas (CSAs) occur when there is a transient reduction by the ponto-medullary respiratory rhythm generator. In contrast, obstructive sleep apnea (OSA) involves continuous respiratory efforts made against a closed airway. The four major types of sleep apnea are depicted in Figure 1.

  4. 30 maj 2024 · OSA can lead to dynamic fluctuations in heart rate during sleep, characterized by increases during apnea episodes and arousals, as well as potential episodes of bradycardia.

  5. 28 gru 2018 · The CANPAP (Canadian Continuous Positive Airway Pressure for Patients with Central Sleep Apnea and Heart Failure) trial investigated the effect of CPAP treatment in patients with HF and CSA.

  6. 28 lip 2008 · An 11-year follow-up of older residents in San Diego (Calif) showed the mortality rate for cardiovascular disease to be higher for those with OSA (35% for AHI <15, 56% for AHI ≥15). 46 Prospective analyses of the Wisconsin Sleep Cohort Study indicate that OSA increases the risk of incident hypertension. 56 Snoring, as a marker of OSA ...

  7. Abstract. Sleep apnea is highly prevalent in patients with cardiovascular disease. These disordered breathing events are associated with a profile of perturbations that include intermittent hypoxia, oxidative stress, sympathetic activation, and endothelial dysfunction, all of which are critical mediators of cardiovascular disease.