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6 maj 2022 · In addition to glycolysis and oxidative phosphorylation (OXPHOS), the oxidative part of the pentose phosphate pathway (PPP) plays an important role during the cell cycle by providing reduced NADPH for the redox system, thus lowering the risk of generating mutations during DNA replication.
- The bidirectional relationship between metabolism and cell cycle ...
Cells must rewire metabolism to meet changing biosynthetic...
- The bidirectional relationship between metabolism and cell cycle ...
Cells couple the exergonic reaction of ATP hydrolysis to whatever work needs to be done. One example of energy coupling using ATP is the sodium-potassium pump (Na + /K + pump), which drives sodium out of the cell and potassium into the cell.
13 lut 2023 · In addition to providing energy, the breakdown of ATP through hydrolysis serves a broad range of cell functions, including signaling and DNA/RNA synthesis. ATP synthesis utilizes energy obtained from multiple catabolic mechanisms, including cellular respiration, beta-oxidation, and ketosis.
These organelles replicate by dividing in two, using a process similar to the simple, asexual form of cell division employed by bacteria. Video microscopy shows that mitochondria are incredibly...
14 gru 2018 · This Primer explores how a systematic understanding of cellular ATP regulation and function at the single-cell level is now becoming possible with the help of CRISPRi-based screening and genetically-encoded ATP biosensors.
27 cze 2023 · Cells must rewire metabolism to meet changing biosynthetic demands across cell cycle phases. In turn, metabolism can influence cell cycle progression through direct regulation of cell cycle proteins, through nutrient-sensing signaling pathways, and through its impact on cell growth, which is linked to cell division.
16 sie 2023 · Replisomes assemble at specific replication initiation sites known as origins, from where they initiate and carry out DNA synthesis. To ensure that chromosomes replicate once per cell cycle, overlapping regulatory mechanisms tightly control the initiation phase of replication and prevent unscheduled re-initiation events [1–3].
