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  1. 15 wrz 2022 · Example of an LV apical mural (laminar) thrombus (red arrows) seen on gadolinium-enhanced CMR. Note the adjacent white appearing areas indicating infarcted myocardium. CMR indicates cardiac magnetic resonance; and LV, left ventricular.

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  2. 5 mar 2024 · Left ventricular (LV) thrombus may develop after acute myocardial infarction (MI) and occurs most often with a large, anterior ST-elevation MI (STEMI). However, the use of reperfusion therapies, including percutaneous coronary intervention and fibrinolysis, has significantly reduced the risk.

  3. Left ventricular (LV) thrombus is a feared complication of LV dysfunction associated with high rates of systemic embolism, morbidity, and mortality. Traditionally, LV thrombus has been associated with acute myocardial infarction (MI).

  4. These embolic complications could occur at any time under antithrombotic therapy, as a consequence of thrombus fragmentation, quick thrombus regression, or on the contrary, persistence of the thrombus.

  5. Causes of de novo development of intra-cardiac thrombi include atrial brillation,6 valvular disease,2,7 fi stasis within the left atrial appendage,8 lead/device-associated thrombi9,10 and predisposition to development of mural thrombi — such as advanced systolic heart failure (HF),11dilated cardiomy-opathy,12 acute myocardial infarction (MI)13an...

  6. The management of patients with left ventricular mural thrombus after AMI is challenging because insufficient evidence is available from randomized trials. • In patients with or at high risk of developing LV thrombus, serial imaging may identify features warranting more intensive or prolonged antithrombotic therapy to reduce the risk of embolism.

  7. 4 lip 2023 · It is vital to treat mural thrombi as this can lower the risk of stroke, myocardial infarction, and pulmonary embolism. There are no standardized guidelines for the treatment of mural thrombi. The type of treatment depends on the location of thrombi, patient symptoms, and urgency of the situation.

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