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15 wrz 2022 · Example of an LV apical mural (laminar) thrombus (red arrows) seen on gadolinium-enhanced CMR. Note the adjacent white appearing areas indicating infarcted myocardium. CMR indicates cardiac magnetic resonance; and LV, left ventricular.
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- Linda S. Williams
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Background—The Randomized Evaluation of Long-term...
- Results From the GEIST
Left ventricular (LV) thrombus formation is a well‐known...
- Intraventricular Thrombus Formation and Embolism in Takotsubo Syndrome
Objective: Takotsubo syndrome (TTS) is characterized by...
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- Peripartum Cardiomyopathy
Peripartum cardiomyopathy is a potentially life-threatening...
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Introduction. The advent of reperfusion therapy and the widespread use of primary percutaneous coronary intervention (PCI) have markedly reduced the incidence of post–myocardial infarction (MI) left ventricular thrombus (LVT) over the last decades (1–3).
14 wrz 2022 · We suggest that, on the basis of limited data, it may be prudent to treat patients with OAC for newly diagnosed mural (laminated) LV thrombus as one would a patient with a protruding or mobile thrombus.
Introduction. Left ventricular (LV) thrombus is a feared complication of LV dysfunction associated with high rates of systemic embolism, morbidity, and mortality. Traditionally, LV thrombus has been associated with acute myocardial infarction (MI).
The 2013 American College of Cardiology Foundation/AHA STEMI guidelines recommend as reasonable (Class 2a, Level of Evidence: C) 3 months of VKA therapy for patients with STEMI and asymptomatic LV mural thrombus with a target INR of 2.0-2.5 when combining OAC with DAPT. 5 The 2014 AHA/American Stroke Association stroke guidelines recommend 3 ...
Daniel Lenihan, MD. Abstract: Intracardiac thrombus is a condition of increasing clinical significance not only because of its potential complications but also because of the lack of clinical evidence to guide clinicians in selecting optimal therapies.
Abstract. Introduction Left ventricular thrombus (LVT) is a frequent complication of left ventricular systolic dysfunction (1). Incidence following acute myocardial infarction is estimated at 13–20% and up to 15% in with non-ischaemic cardiomyopathy (2, 3). Once diagnosed, guidelines recommend anticoagulation with vitamin K antagonists (VKA ...