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15 wrz 2022 · Example of an LV apical mural (laminar) thrombus (red arrows) seen on gadolinium-enhanced CMR. Note the adjacent white appearing areas indicating infarcted myocardium. CMR indicates cardiac magnetic resonance; and LV, left ventricular.
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- Linda S. Williams
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Background—The Randomized Evaluation of Long-term...
- Results From the GEIST
Left ventricular (LV) thrombus formation is a well‐known...
- Intraventricular Thrombus Formation and Embolism in Takotsubo Syndrome
Objective: Takotsubo syndrome (TTS) is characterized by...
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- Peripartum Cardiomyopathy
Peripartum cardiomyopathy is a potentially life-threatening...
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A calcified LVT was defined as a persistent left ventricular mural thrombus encapsulated by thickened and calcified endocardium. Left ventricular characteristics including left ventricular ejection fraction (LVEF), left ventricular volume, wall motion, cardiac output, and potential mechanical complications were also collected.
Current treatment strategies for LV thrombus, including direct oral anticoagulant (DOAC) use, are also unclear and require examination . We identified patients with LV thrombus on echocardiography (with and without contrast) at Brigham and Women’s Hospital between January 2008 and May 2015.
23 wrz 2022 · On the basis of limited data, patients with nonischemic cardiomyopathy with LV thrombus should be treated with OAC for at least 3–6 months, with discontinuation if LV ejection fraction improves to >35% (assuming resolution of the LV thrombus) or if major bleeding occurs.
This analysis supports the hypotheses that 1) mural thrombus after myocardial infarction poses a significantly increased risk of embolization, 2) the risk of embolization is reduced by systemic anticoagulation, and 3) anticoagulation can prevent mural thrombus formation.
The most likely explanations for a mural thrombus in the LV are severe LV dysfunction and/or recent anterior wall MI. Echocardiography is the primary modality to detect LVT.
7 kwi 2020 · The authors identified 159 patients with confirmed LV thrombus. These patients were treated with vitamin K antagonists (48.4%), parenteral heparin (27.7%), or direct oral anticoagulants (22.6%). Antiplatelet therapy was used in 67.9% of cases.