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15 wrz 2022 · Example of an LV apical mural (laminar) thrombus (red arrows) seen on gadolinium-enhanced CMR. Note the adjacent white appearing areas indicating infarcted myocardium. CMR indicates cardiac magnetic resonance; and LV, left ventricular.
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- Linda S. Williams
Specific recommendations for prevention strategies often...
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Background—The Randomized Evaluation of Long-term...
- Results From the GEIST
Left ventricular (LV) thrombus formation is a well‐known...
- Intraventricular Thrombus Formation and Embolism in Takotsubo Syndrome
Objective: Takotsubo syndrome (TTS) is characterized by...
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4 lip 2023 · Mural thrombus occurrence in a normal or minimally atherosclerotic vessel is a rare entity in the absence of a hypercoagulative state or inflammatory, infectious, or familial aortic ailments. Mural thrombi can be seen in large vessels such as the heart and aorta and can restrict blood flow.
For the purpose of this paper our definition of an apical mural thrombus is a distinct mass of echoes, most commonly seen in the apex throughout the cardiac cycle, and in more than one view. Mural thrombi are most commonly seen between six and 10 days following an acute myocardial infarction (MI).
The diagnosis of left ventricular thrombus is of potential clinical importance in identifying patients at risk for systemic embolization. Over the past years, several sophisticated methods capable of detecting thrombus have been developed.
Apical four chamber view in a patient with a bioprosthetic mitral valve and spontaneous echo contrast seen in the left ventricle. The swirling of red blood cells in the sequence A-C is often seen in low cardiac output states and requires careful investigation for an underlying mural thrombus.
12 gru 2022 · Left ventricular (LV) apical thrombus formation is a well described and clinically important complication of acute myocardial infarction (MI) with a substantial risk of thromboembolism. Alterations in the inflammatory status may contribute to this complication.
The differential diagnosis of apical HCM includes mural thrombus, hypertrabeculation or non-compaction and endomyocardial fibrosis. These entities may be diagnosed on MRI using steady-state free precession (SSFP) imaging techniques and LGE imaging.
