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Digoxin ECG changes: arrhythmias, conduction defects and waveform changes. Digoxin may be used in patients with heart failure, atrial fibrillation, atrial flutter, and in selected cases of paroxysmal supraventricular tachycardia.
- Atrioventricular
Digoxin: Recall that digoxin may cause all arrhythmias and...
- Atrioventricular
8 paź 2024 · Digoxin effect refers to the presence on the ECG of: Downsloping ST depression with a characteristic “reverse tick” or “Salvador Dali sagging” appearance; Flattened, inverted, or biphasic T waves; Shortened QT interval
8 paź 2024 · ECG Features of Digoxin Toxicity. The classic digoxin toxic dysrhythmia combines: Supraventricular tachycardia (due to increased automaticity) Slow ventricular response (due to decreased AV conduction) Other common dysrhythmias associated with digoxin toxicity include:
Digoxin toxicity is characterised by gastrointestinal distress, hyperkalemia and life-threatening dysryhthmias, including increased automaticity and AV nodal blockade; Digoxin has a narrow therapeutic index and chronic toxicity is more likely in the elderly and those with renal impairment
19 sty 2023 · The kidneys excrete digoxin in direct proportion to the glomerular filtration rate; the liver metabolizes 16%. Digoxin has a half-life that varies from 36 to 48 hours, which may increase in cases of renal failure.
The diagnosis of digoxin toxicity is based primarily on clinical suspicion and clinical features (including ECG changes) suggestive of digoxin intoxication, and can be confirmed by assessment of serum digoxin levels.
8 sie 2024 · At therapeutic digoxin concentrations (0.6 to 1.2 nanomol/L [0.5 to 0.9 nanograms/mL]), ECG may show PR-interval prolongation and a scooped ST segment. With toxicity, ECG may show signs of increased automaticity (premature ventricular contractions), atrioventricular nodal blockade, and slowed ventricular response.
